https://ogma.newcastle.edu.au/vital/access/ /manager/Index ${session.getAttribute("locale")} 5 https://ogma.newcastle.edu.au/vital/access/ /manager/Repository/uon:38040 Wed 28 Jul 2021 10:17:50 AEST ]]> https://ogma.newcastle.edu.au/vital/access/ /manager/Repository/uon:36258 Wed 09 Feb 2022 15:58:15 AEDT ]]> https://ogma.newcastle.edu.au/vital/access/ /manager/Repository/uon:49006 Wed 03 May 2023 12:17:18 AEST ]]> https://ogma.newcastle.edu.au/vital/access/ /manager/Repository/uon:21293 Sat 24 Mar 2018 07:54:36 AEDT ]]> https://ogma.newcastle.edu.au/vital/access/ /manager/Repository/uon:20655 Sat 24 Mar 2018 07:49:58 AEDT ]]> https://ogma.newcastle.edu.au/vital/access/ /manager/Repository/uon:39241 P = 0.022 adjusted for age, sex, and smoking). Cecal IELs were increased in IBS—diarrhea (relative risk ratio = 2.03, 95% confidence interval 1.13–3.63, P = 0.017). No difference was observed in alpha diversity of MaM or fecal microbiota based on IEL count. There was no difference in beta diversity of the MaM according to IEL count in the terminal ileal (TI) (P = 0.079). High TI IEL counts associated with a significant expansion of the genus Blautia (P = 0.024) and unclassified Clostridiales (P = 0.036) in colon MaM. Discussion: A modest but significant increase in IELs was observed in IBS vs. controls in a population-based setting. Subtle TI and cecal inflammation may play a pathogenic role in IBS but needs confirmation. Modest but discernible differences in the colonic MaM were seen according to TI IEL count but not IBS status.]]> Fri 27 May 2022 14:39:27 AEST ]]>